NIH Weekly Funding Opportunities and Policy Notices
Notice NOT-OD-20-052 from the NIH Guide for Grants and Contracts
Notice NOT-HL-19-740 from the NIH Guide for Grants and Contracts
Notice NOT-HL-19-736 from the NIH Guide for Grants and Contracts
Notice NOT-DA-20-028 from the NIH Guide for Grants and Contracts
Notice NOT-AI-20-022 from the NIH Guide for Grants and Contracts
Notice NOT-NS-20-022 from the NIH Guide for Grants and Contracts
Notice NOT-OD-20-045 from the NIH Guide for Grants and Contracts
Notice NOT-NS-20-021 from the NIH Guide for Grants and Contracts
Notice NOT-TW-19-008 from the NIH Guide for Grants and Contracts
Notice NOT-OD-20-024 from the NIH Guide for Grants and Contracts
Notice NOT-OD-20-025 from the NIH Guide for Grants and Contracts
Notice NOT-OD-20-023 from the NIH Guide for Grants and Contracts
Notice NOT-AI-20-018 from the NIH Guide for Grants and Contracts
Notice NOT-HL-19-730 from the NIH Guide for Grants and Contracts
Notice NOT-OD-20-017 from the NIH Guide for Grants and Contracts
Funding Opportunity RFA-AG-21-002 from the NIH Guide for Grants and Contracts. This FOA will support research on aspects of rejuvenation and accelerated aging observed specifically in heterochronic blood exchange (HBE) experiments. The objectives are to identify the multiple factors involved, the multiple cell types involved and the mechanisms underlying rejuvenation or accelerated aging that is observed in the transfer of phenotypes between young and old laboratory animals. It is also anticipated that molecular signatures of rejuvenation or accelerated aging will be obtained from research supported under this FOA.
Funding Opportunity RFA-AG-21-010 from the NIH Guide for Grants and Contracts. Recent reports highlight the enormous spatial and temporal diversity of glia, even within the same glial cell type. This within-glial-cell-type heterogeneity evolves during aging, suggesting that subtypes of glia with distinct physiological roles could emerge to influence brain aging processes. The goal of this Funding Opportunity Announcement is to support research addressing critical knowledge gaps in our understanding of how these glial subpopulations could contribute to vulnerability and resilience to brain aging.
Funding Opportunity RFA-HD-21-013 from the NIH Guide for Grants and Contracts. The purpose of this funding opportunity announcement (FOA) is to support innovative, multidisciplinary, interactive, and synergistic program projects that integrate basic, translational, and clinical approaches to understanding the developmental biology and genetic basis of significant congenital human malformations. To contain costs, each program project will consist of only three component research projects, as well as associated cores. At least one project must use basic research in an animal model system and at least one project must be clinical or translational in nature. The component research projects must share a common central theme, focus, or objective on a specific major developmental defect or malformation that is genotypically, mechanistically, biologically, or phenotypically analogous or homologous in both animal models and humans. Any non-mammalian or mammalian animal model may be used, as long as it contributes to the common overall theme or objective of the program project. The component research projects should share a common developmental gene, process, mechanism, pathway, or phenotype.
Notice NOT-AG-20-010 from the NIH Guide for Grants and Contracts
Notice NOT-AI-20-013 from the NIH Guide for Grants and Contracts